globalchange  > 气候变化事实与影响
DOI: 10.1289/ehp.1206458
论文题名:
Epigenetic Influences on Associations between Air Pollutants and Lung Function in Elderly Men: The Normative Aging Study
作者: Johanna Lepeule; 1 Marie-Abele Catherine Bind; 1; rea A. Baccarelli; 1 Petros Koutrakis; 1 Letizia Tarantini; 2 Augusto Litonjua; 3; 4 David Sparrow; 5 Pantel Vokonas; 5; Joel D. Schwartz1; 3
刊名: Environmental Health Perspectives
ISSN: 0091-7447
出版年: 2014
卷: Volume 122, 期:Issue 6
起始页码: 566
语种: 英语
英文摘要: Background: Few studies have been performed on pulmonary effects of air pollution in the elderly—a vulnerable population with low reserve capacity—and mechanisms and susceptibility factors for potential effects are unclear.

Objectives: We evaluated the lag structure of air pollutant associations with lung function and potential effect modification by DNA methylation (< or ≥ median) at 26 individual CpG sites in nine candidate genes in a well-characterized cohort of elderly men.

Methods: We measured forced vital capacity (FVC), forced expiratory volume in 1 sec (FEV1), and blood DNA methylation one to four times between 1999 and 2009 in 776 men from the Normative Aging Study. Air pollution was measured at fixed monitors 4 hr to 28 days before lung function tests. We used linear mixed-effects models to estimate the main effects of air pollutants and effect modification by DNA methylation.

Results: An interquartile range (IQR) increase in subchronic exposure (3 to 28 days cumulated), but not in acute exposure (during the previous 4 hr, or the current or previous day), to black carbon, total and nontraffic particles with aerodynamic diameter ≤ 2.5 μm (PM2.5), carbon monoxide, and nitrogen dioxide was associated with a 1–5% decrease in FVC and FEV1 (p < 0.05). Slope estimates were greater for FVC than FEV1, and increased with cumulative exposure. The estimates slopes for air pollutants (28 days cumulated) were higher in participants with low (< median) methylation in TLR2 at position 2 and position 5 and high (≥ median) methylation in GCR.

Conclusions: Subchronic exposure to traffic-related pollutants was associated with significantly reduced lung function in the elderly; nontraffic pollutants (particles, ozone) had weaker associations. Epigenetic mechanisms related to inflammation and immunity may influence these associations.
URL: https://ehp.niehs.nih.gov/1206458
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资源类型: 期刊论文
标识符: http://119.78.100.158/handle/2HF3EXSE/12776
Appears in Collections:气候变化事实与影响
气候变化与战略

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作者单位: 1Exposure, Epidemiology, and Risk Program, Department of Environmental Health, Harvard School of Public Health, Boston, Massachusetts, USA; 2Center of Molecular and Genetic Epidemiology, Department of Environmental and Occupational Health, Ca’ Granda Ospedale Maggiore Policlinico IRCCS Foundation, Università degli Studi di Milano, Milan, Italy; 3Channing Laboratory, and 4Division of Pulmonary and Critical Care Medicine, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA; 5VA Normative Aging Study, Veterans Affairs Boston Healthcare System and the Department of Medicine, Boston University School of Medicine, Boston, Massachusetts, USA

Recommended Citation:
Johanna Lepeule,1 Marie-Abele Catherine Bind,1,et al. Epigenetic Influences on Associations between Air Pollutants and Lung Function in Elderly Men: The Normative Aging Study[J]. Environmental Health Perspectives,2014-01-01,Volume 122(Issue 6):566
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