globalchange  > 气候变化与战略
DOI: 10.1073/pnas.1702984114
论文题名:
Phosphorylation and negative regulation of CONSTITUTIVELY PHOTOMORPHOGENIC 1 by PINOID in Arabidopsis
作者: Lin F.; Xu D.; Jiang Y.; Chen H.; Fan L.; Holm M.; Deng X.W.
刊名: Proceedings of the National Academy of Sciences of the United States of America
ISSN: 0027-8424
出版年: 2017
卷: 114, 期:25
起始页码: 6617
结束页码: 6622
语种: 英语
英文关键词: Arabidopsis ; COP1 ; Phosphorylation ; Photomorphogenesis ; PID
Scopus关键词: constitutively photomorphogenic 1 protein ; plant protein ; unclassified drug ; Arabidopsis protein ; COP1 protein, Arabidopsis ; PINOID protein, Arabidopsis ; protein binding ; protein serine threonine kinase ; ubiquitin protein ligase ; adult ; Arabidopsis ; Article ; enzyme activity ; Escherichia coli ; gene interaction ; hypocotyl ; in vitro study ; nonhuman ; phenotype ; photomorphogenesis ; plant development ; priority journal ; protein degradation ; protein expression ; protein phosphorylation ; protein processing ; protein protein interaction ; regulatory mechanism ; start codon ; stop codon ; Arabidopsis ; gene expression regulation ; light ; metabolism ; phosphorylation ; physiology ; ubiquitination ; Arabidopsis ; Arabidopsis Proteins ; Gene Expression Regulation, Plant ; Light ; Phosphorylation ; Protein Binding ; Protein Processing, Post-Translational ; Protein-Serine-Threonine Kinases ; Ubiquitin-Protein Ligases ; Ubiquitination
英文摘要: CONSTITUTIVELY PHOTOMORPHOGENIC 1 (COP1) plays crucial roles in various cellular processes via its E3 ubiquitin ligase activity in organisms, ranging from fungi to humans. As a key component in regulating various biological events, COP1 itself is precisely controlled at multiple layers. Here, we report a negative regulator of COP1, PINOID (PID), which positively mediates photomorphogenic development. Specifically, PID genetically and physically interacts with COP1 and directly phosphorylates COP1 at Ser20. As a result, this posttranslational modification serves to repress COP1 activity and promote photomorphogenesis. Our findings signify a key regulatory mechanism for precisely maintaining COP1 activity, thereby ensuring appropriate development in plants.
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资源类型: 期刊论文
标识符: http://119.78.100.158/handle/2HF3EXSE/163828
Appears in Collections:气候变化与战略

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作者单位: Lin, F., Department of Biology, Southern University of Science and Technology, Shenzhen, 518055, China, State Key Laboratory of Protein and Plant Gene Research, Peking-Tsinghua Center for Life Sciences, School of Advanced Agriculture Sciences, School of Life Sciences, Peking University, Beijing, 100871, China; Xu, D., Department of Biology, Southern University of Science and Technology, Shenzhen, 518055, China; Jiang, Y., Department of Biology, Southern University of Science and Technology, Shenzhen, 518055, China, State Key Laboratory of Protein and Plant Gene Research, Peking-Tsinghua Center for Life Sciences, School of Advanced Agriculture Sciences, School of Life Sciences, Peking University, Beijing, 100871, China; Chen, H., State Key Laboratory of Protein and Plant Gene Research, Peking-Tsinghua Center for Life Sciences, School of Advanced Agriculture Sciences, School of Life Sciences, Peking University, Beijing, 100871, China; Fan, L., State Key Laboratory of Protein and Plant Gene Research, Peking-Tsinghua Center for Life Sciences, School of Advanced Agriculture Sciences, School of Life Sciences, Peking University, Beijing, 100871, China; Holm, M., Department of Biological and Environmental Sciences, Gothenburg University, Gothenburg, SE-405 30, Sweden; Deng, X.W., Department of Biology, Southern University of Science and Technology, Shenzhen, 518055, China, State Key Laboratory of Protein and Plant Gene Research, Peking-Tsinghua Center for Life Sciences, School of Advanced Agriculture Sciences, School of Life Sciences, Peking University, Beijing, 100871, China

Recommended Citation:
Lin F.,Xu D.,Jiang Y.,et al. Phosphorylation and negative regulation of CONSTITUTIVELY PHOTOMORPHOGENIC 1 by PINOID in Arabidopsis[J]. Proceedings of the National Academy of Sciences of the United States of America,2017-01-01,114(25)
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