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DOI: 10.1371/journal.pone.0121842
论文题名:
Diacerein Improves Left Ventricular Remodeling and Cardiac Function by Reducing the Inflammatory Response after Myocardial Infarction
作者: Anali Galluce Torina; Karla Reichert; Fany Lima; Karlos Alexandre de Souza Vilarinho; Pedro Paulo Martins de Oliveira; Helison Rafael Pereira do Carmo; Daniela Diógenes de Carvalho; Mário José Abdalla Saad; Andrei Carvalho Sposito; Orlando Petrucci
刊名: PLOS ONE
ISSN: 1932-6203
出版年: 2015
发表日期: 2015-3-27
卷: 10, 期:3
语种: 英语
英文关键词: Myocardial infarction ; Fibrosis ; Gene expression ; Inflammation ; Hemodynamics ; Remote areas ; Collagens ; Saline solutions
英文摘要: Background The inflammatory response has been implicated in the pathogenesis of left ventricular (LV) remodeling after myocardial infarction (MI). An anthraquinone compound with anti-inflammatory properties, diacerein inhibits the synthesis and activity of pro-inflammatory cytokines, such as tumor necrosis factor and interleukins 1 and 6. The purpose of this study was to investigate the effects of diacerein on ventricular remodeling in vivo. Methods and Results Ligation of the left anterior descending artery was used to induce MI in an experimental rat model. Rats were divided into two groups: a control group that received saline solution (n = 16) and a group that received diacerein (80 mg/kg) daily (n = 10). After 4 weeks, the LV volume, cellular signaling, caspase 3 activity, and nuclear factor kappa B (NF-κB) transcription were compared between the two groups. After 4 weeks, end-diastolic and end-systolic LV volumes were reduced in the treatment group compared to the control group (p < .01 and p < .01, respectively). Compared to control rats, diacerein-treated rats exhibited less fibrosis in the LV (14.65%± 7.27% vs. 22.57%± 8.94%; p < .01), lower levels of caspase-3 activity, and lower levels of NF-κB p65 transcription. Conclusions Treatment with diacerein once a day for 4 weeks after MI improved ventricular remodeling by promoting lower end-systolic and end-diastolic LV volumes. Diacerein also reduced fibrosis in the LV. These effects might be associated with partial blockage of the NF-κB pathway.
URL: http://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0121842&type=printable
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资源类型: 期刊论文
标识符: http://119.78.100.158/handle/2HF3EXSE/21374
Appears in Collections:过去全球变化的重建
影响、适应和脆弱性
科学计划与规划
气候变化与战略
全球变化的国际研究计划
气候减缓与适应
气候变化事实与影响

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作者单位: Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Department of Surgery, Discipline of Cardiac Surgery, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Department of Internal Medicine, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Department of Internal Medicine, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Laboratory of Myocardial Ischemia/Reperfusion, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil;Department of Surgery, Discipline of Cardiac Surgery, Faculty of Medical Science, State University of Campinas—UNICAMP, Campinas, SP, Brazil

Recommended Citation:
Anali Galluce Torina,Karla Reichert,Fany Lima,et al. Diacerein Improves Left Ventricular Remodeling and Cardiac Function by Reducing the Inflammatory Response after Myocardial Infarction[J]. PLOS ONE,2015-01-01,10(3)
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